EMF Blocking in addition to EMF Shielding

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EMF Blocking in addition to EMF Shielding

No matter if you live in a house or apartment or just need to ensure that your house is free of electromagnetic fields, there are a number options to limit your exposure. One of the easiest is to restrict the use of electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from entering your home. Another method to protect your house from EMF radiations is to use a shielding canopy for RF. This is a fabric made of net that has EMF shielding and is used to stop EMFs from entering rooms. Another option is to have your house equipped with an electrical enclosure. These devices are called Faraday cages.


A number of studies have proven how the non-ionizing energy of RF can cause antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to be based on the downregulation the cancer stem cell. This could explain the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM EMF's effects on cancer patients is not clear.

Effects from AM RF EMF on HCC tumour growth in vivo were examined in mice. The tumors were split into 3 groups. First, the group that was unaffected to RF EMF. The second group was exposed to RF EMF at frequencies similar to that used in humans. In the third, they were subjected RF EMF with HCC-specific modulation frequencies. The impact of HCCMF on tumours was assessed against the effect of RCF.  block emf  revealed that tumors treated by HCCMF had significant shrinkage. However, the tumors treated with RCF didn't show evidence of shrinkage in the tumour.

The mechanism of tumour-specific AM RF EMF could be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effect upon HCC cells is caused by CACNA1H the protein which regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have more broader implications for the treatment and diagnosis of many cancers.

The tumours in the control group were not exposed EMF from RF, and fed a normal diet of mice. The tumors of those in the HCCMF group were treated with Huh7 cells when they were five-seven weeks old. The tumours were then euthanized when they showed excessive burden.

The tumours from the three groups also displayed different growth curves.  emf blockers  treated with HCCMF had a significant reduction in the size of the tumour after eight weeks. However, tumors that were treated using RCF showed no reduction in size. The difference was highly significant. The tumours treated with RCF had necrosis, which is common in tumours exposed to RCF. There is a possibility that the necrosis is caused by a lack of oxygen in the more invasive cancers.

In  You can find out more , the results show the fact that AM EMF has anticancer properties in vitro as well as in vivo. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors in HCC patients. It is possible that AM RF EMF causes these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. Furthermore, AM RF EMF may have a long-lasting influence on the development of HCC tumours in vivo.