If you reside in a house or apartment or just want to keep your home free from EMFs There are a variety options to limit your exposure. One of the easiest is to limit the use of electronic devices. https://ctxt.io/2/AACQPi3YFg could also consider EMF block paint to stop EMF radiation from reaching your home. Another method to protect your home against EMF radiation is to use an RF shielding canopy. This is a fabric made of net that has EMF shielding and is used to stop EMFs from entering a room. Another option is to have your home equipped with an electrical enclosure. These devices are called Faraday cages.
Numerous studies have demonstrated that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation in cancer-related stem cells. This may account for the long-term responses seen in some patients with advanced HCC. However, the mechanism behind AM EMF's effects on patients with cancer is not evident.
Effects from AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumors were split into 3 groups. The first group was not exposed to RF EMF. Second group members were exposed to RF EMF at the same frequency to the frequency used by humans. Third group members were exposed to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. emf blocking showed that the tumours treated with HCCMF were significantly shrinking. However, emf blocker treated with RCF didn't show evidence of tumour shrinkage.
The reason for cancer-specific AM RF EMF may be based on the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is controlled by CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The findings suggest that CACNA1H may have broader implications for diagnosis and treatment of various cancers.
The tumours in the control group were not exposed EMF from radiofrequency, and fed a normal diet of mice. The tumors in HCCMF HCCMF group were injected with Huh7 cells when they were 5 to 7 weeks old. The tumors were then killed after they had a high burden.
The tumours in the three groups showed distinct growth curves. The HCCMF-treated tumors saw a significant reduction in size of the tumor after eight weeks. However, the tumors which were treated by RCF didn't show shrinkage. The difference was significant. The tumors treated with RCF had necrosis, which is common in tumours exposed to RCF. There is a possibility that the necrosis is caused by an absence of oxygen in the larger cancers.
In conclusion, the findings suggest an AM-RF EMF has anticancer activity in vitro and in vivo. Several studies have shown the fact that AM RF EMF produces measurable tumour shrinkage in HCC patients. There is a possibility that AM RF EMF causes these effects through CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may cause a lasting effect on the growth of HCC tumours in living tissue.