If you reside in a house or apartment, or you just want to keep your home free of electromagnetic fields T here are plenty of methods to reduce exposure. https://shea-chung-2.thoughtlanes.net/emf-block-paint-in-addition-to-emf-shielding-1680932134 of the easiest is to restrict the use of electronic devices. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another way to shield your house from EMF radiation would be to use a RF shielding canopy. It is a type made of net that has EMF shielding and is used to block EMFs from entering a room. Another alternative is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown studies have shown that nonionizing EMF produces antiproliferative effects in HCC cells. The mechanism that drives AM RF EMF's anticancer activity in vitro is believed to result from the deregulation of cancer stem cells. This could explain the long-term responses seen in certain patients suffering from advanced HCC. But, the reason for AM RF EMF's effect in patients with cancer is not yet clear.
The effects on the effects of AM RF EMFs on HCC tumor growth in vivo were studied in mice. The tumours were divided into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected to RF EMF at the same frequency to that used in humans. The third group was exposed to RF EMF with HCC-specific modulation frequencies. The effect of HCCMF on the tumours was evaluated against that of RCF. The results indicated that cancers treated with HCCMF had significant shrinkage. However, tumours treated with RCF didn't show evidence of shrinkage of the tumor.
The reason for cancer-specific AM RF EMF might be based on the fact that cancer cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is caused by CACNA1H, a protein which is responsible for the influx of Ca2+ specific to tumours. The results suggest that CACNA1H could have wider implications for diagnosis and treatment of various cancers.
The tumors in the controls were never exposed RF EMF, and were fed a normal mouse diet. The tumours in the HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized after they had a high burden.
The tumours from the three groups showed distinct growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after eight weeks. However, the tumours treated with RCF showed no signs of shrinkage. The difference was substantial. The tumors treated with RCF were able to show necrosis that is common when tumors are that are exposed to RCF. It is possible that the necrosis was caused by an absence of oxygen in larger cancers.
In emf radiation blocker , the results suggest an AM-RF EMF has anticancer effects in vitro and in the vivo. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. The possibility is that the AM EMF causes these effects through CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the development of HCC tumors in living tissue.