If you reside in a house or apartment, or you just would like to keep your home free of EMFs, there are a number options to limit your exposure. emf blocking of the easiest is to reduce the usage of your electronic devices. It is also possible to use EMF blocker paint to block EMF radiation from entering your house. Another way to shield your house against EMF radiations is to use a RF shielding canopy. It's a kind of net that contains EMF shielding. It is utilized to block EMFs from entering a room. Another alternative is to have your home equipped with a conductive enclosure. These devices are called Faraday cages.
Several studies have shown that the non-ionizing RF EMF has antiproliferative effects in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is thought to be based on the downregulation of cancer stem cells. This could explain the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM EMF's impact on cancer patients is not clear.
Effects on the effects of AM electromagnetic fields (RFEM) on HCC tumor growth in vivo were studied in mice. The tumors were divided into 3 groups. First, the group that was unaffected to RF EMF. Another group of participants was subjected RF EMF at frequencies similar to the frequency used by humans. In the third, they were subjected to the RF EMF in HCC-specific frequencies. The impact of HCCMF on the tumours was compared to that of RCF. The results indicated that tumours treated with HCCMF were significantly shrinking. However, the tumours treated with RCF showed no evidence of tumour shrinkage.
emf blocking for tumour-specific AM RF EMF might be based on the fact that tumor cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is mediated through CACNA1H which is a protein that mediates tumour-specific Ca2+ influx. The findings suggest that CACNA1H could have more broader implications for diagnosis and treatment of a variety of cancers.
The tumours in the controls were never exposed to EMF from RF, and fed a normal mouse diet. The tumours in the HCCMF group were injected with Huh7 cells after they were five-seven weeks old. The tumours were then euthanized after they had a high burden.
The tumors in the three groups also showed distinct growth curves. The tumors treated with HCCMF showed a significant decrease in tumour size after eight weeks. However, emf blocker treated with RCF showed no reduction in size. The difference was highly significant. The tumors treated with RCF showed necrosis, which is typical when tumors are exposed to RCF. It is possible that this necrosis was caused by the lack of oxygen in larger tumors.
In summary, the results show that AM RF EMF exhibits anticancer effects in vitro and in the vivo. Several studies have shown that AM RF EMF produces measurable shrinkage of tumors for HCC patients. The possibility is that the AM EMF triggers these effects through CACNA1H which is a protein involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting effect on the development of HCC tumours in living tissue.