If you reside in an apartment or house or just want to keep your home free from EMFs There are plenty of methods to reduce exposure. One of the most effective is to reduce the usage of your electronic devices. It is also possible to use EMF block paint to prevent EMF radiation from reaching your house. Another way to shield your house against EMF radiations is to install a RF shielding canopy. It is a type of net that contains EMF shielding. It's used to block EMFs from entering a room. Another alternative is to have your home equipped with a conductive enclosure. These devices are called Faraday cages.
A number of studies have proven that the non-ionizing RF EMF has anti-proliferative effects on HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed result from the deregulation in cancer-related stem cells. This could be the reason for the long-term responses seen in patients with advanced HCC. But, the reason for AM EMF's effects on cancer patients is not evident.
emf blockers from AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumours were separated in three different groups. First, the group that was unaffected to RF EMF. emf blocking were exposed RF EMF at the same frequency to the frequency used by humans. The third group was exposed to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that cancers treated with HCCMF had significant shrinkage. However, tumours treated with RCF did not show any evidence of shrinkage in the tumour.
The mechanism of tumour-specific AM RF EMF may be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is mediated through CACNA1H which is a protein which regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have more broader implications in the treatment and diagnosis of various cancers.
The tumours of those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumours in the HCCMF group were injected with Huh7 cells at the time they were between five and seven weeks old. The tumours were then euthanized in cases of excessive burden.
The tumors of the three groups also displayed distinct growth curves. emf blockers treated with HCCMF saw a significant decrease in size of the tumor after 8 weeks. However, the tumours that were treated using RCF showed no shrinkage. The difference was highly significant. The tumors treated by RCF showed necrosis, which is typical in tumors that have been exposed to RCF. It is possible that this necrosis was caused by a lack of oxygen in the larger tumors.
In sum, the results suggest that AM RF EMF exhibits anticancer effects in vitro and in live. Numerous studies have demonstrated the fact that AM RF EMF produces measurable reduction in tumours within HCC patients. The possibility is that the AM EMF triggers these effects because of CACNA1H, a protein involved in the process of tissue-specific Ca2+ influx. In addition, AM RF EMF may have a long-lasting influence on the growth of HCC tumors in living tissue.