EMF Block Paint and EMF Shielding

· 2 min read
EMF Block Paint and EMF Shielding

If you reside in an apartment or a house, or you just want to keep your home free of EMFs There are plenty options to limit your exposure. One of the most effective is to restrict the usage of your electronic devices. You could also consider EMF block paint to stop EMF radiation from reaching your house. Another method to protect your house from EMF radiations is to put up an RF shielding cover.  phone emf blocker 's a kind made of net which contains EMF shielding. It's used to stop EMFs from entering rooms. Another option is to have your house equipped with an enclosure that is conductive. These enclosures are known as Faraday cages.

Numerous studies have demonstrated that the non-ionizing RF EMF has antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed involve down-regulation the cancer stem cell. This could explain the long-term response seen in certain patients suffering from advanced HCC. However, the reason behind AM RF EMF's effect in patients with cancer is not evident.

Aspects on the effects of AM RF EMFs on HCC tumor growth in vivo was studied in mice. The tumors were classified into three groups. First,  emf neutralizer  that was unaffected to RF EMF. The second group was exposed RF EMF at frequencies similar to that of humans. In the third, they were subjected to RF EMF at HCC-specific modulation frequencies. The effect of HCCMF on the tumours was assessed against the effect of RCF. The results revealed that cancers treated with HCCMF showed significant shrinkage. However, the tumors treated with RCF did not show any evidence of shrinkage of the tumor.



The mechanism of tumour-specific AM RF EMF might be due to the fact that tumour cells require Cav3*2 T-type voltage calcium channels for their proliferation and down-regulation. AM RF EMF's antiproliferative effects in HCC cells is controlled through CACNA1H the protein that regulates the Ca2+ influx specific to tumors. The results suggest that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.

The tumors in those in the group that were unaffected to EMF from RF, and fed a normal mouse diet. The tumors in HCCMF HCCMF group were infected with Huh7 cells after they were 5 to 7 weeks old. The tumours were then euthanized in cases of excessive burden.

The tumors in the three groups also displayed different growth curves. The tumors treated with HCCMF showed a significant decrease in the size of the tumour after 8 weeks. However, the tumors treated with RCF didn't show reduction in size. The difference was significant.  Learn more  treated by RCF were able to show necrosis, which is typical in tumors that have been that are exposed to RCF. It is possible that the necrosis was due to a lack of oxygen in larger tumors.

In conclusion, the findings suggest that AM RF EMF has anticancer activity in vitro and in vivo. A number of studies have proven the fact that AM RF EMF produces measurable reduction in tumours in HCC patients. There is a possibility that AM RF EMF triggers these effects through CACNA1H, a protein that is involved in the tissue-specific Ca2+ influx. Furthermore, AM RF EMF may exert a sustained influence on the growth of HCC tumors in vivo.